Semax is a short synthetic peptide derived from the adrenocorticotropic hormone (ACTH) sequence that has been extensively profiled in vitro and in animal models for neuroprotective and neurotrophic actions. Experimental studies demonstrate Semax influences expression of neurotrophin genes (including BDNF), enhances neuronal survival under ischemic or excitotoxic stress, and modulates cerebral neurotransmitter balance and inflammatory responses in rodent models; mechanistic papers have used gene-expression and biochemical endpoints to delineate Semax’s pathways of action in the central nervous system.

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